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You were once an egg inside your grandmother

Here’s a fact to bring out at your next family dinner: part of you once lived inside your grandmother.

Before your mother was born, her ovaries were already making eggs. One of those eggs would, decades later, become half of you. So while your grandmother was pregnant with your mom, she was also carrying the egg that became you. Three generations, nested like Russian dolls.

That’s just the opening chapter in one of biology’s stranger stories.

The great egg countdown

Most cells in your body are constantly being replaced. Eggs are different: you get your whole supply before you’re born, and that’s it.

The numbers are wild. A female fetus has about 6 to 7 million eggs at 20 weeks of pregnancy. By birth, that’s down to 1 to 2 million. By puberty, somewhere between 300,000 and 500,000 remain1.

How many actually get used? Over a lifetime of periods, only about 400 to 500 eggs are ever released1. The rest, well over 99 percent, quietly retire through a natural process of cell self-destruction. Think of it as a lottery where nearly every ticket expires unused.

The pace of loss picks up in the late thirties. By 50, fewer than 10,000 remain1. When the supply runs low enough, periods stop. That’s menopause.

Your body has brakes

If you have hundreds of thousands of eggs, why don’t you use them all at once? Because your body has a braking system. Each egg sits inside a tiny sac called a follicle, and those follicles make a hormone called anti-Müllerian hormone, or AMH. AMH holds most eggs back, so only a small batch starts developing each month. Without that brake, you could burn through your entire supply in a few cycles1.

AMH levels typically peak in your mid-twenties and fall as the egg supply shrinks1. With a weaker brake, fewer eggs are held back, so a bigger batch starts developing each month. Usually only one is still released; the rest fade away. The countdown to menopause speeds up.

Occasionally more than one egg does get released in the same month. That may be one reason twins are more common in women in their forties1.

It’s the egg, not the uterus

When women in their forties use eggs donated by younger women, their chances of pregnancy are similar to those of younger women1.

In other words, the uterus holds up well with age. The biological clock people talk about is really an egg clock.

Why eggs age: forty-year-old glue

An egg’s chromosomes are held together by proteins called cohesins, a kind of biological glue. That glue is laid down before you’re born and is never replaced. The egg then waits, sometimes for four decades, for its turn to be released4.

Over that time, the glue wears thin. Researchers found that women aged 40 and over had noticeably less of these glue proteins in their eggs than women around 202. Another team found that in human eggs, the paired chromosome copies sit further apart as women get older, a sign the glue is loosening3.

When the glue fails, chromosomes can split the wrong way. Researchers now see this as one of the main reasons eggs are more likely to have the wrong number of chromosomes as we age3,4.

Eggs’ mitochondria, the tiny power plants inside cells, also decline and pick up small errors with age1. Scientists are still working out how much that matters.

Sperm play by different rules

Sperm production starts at puberty and keeps going for life, with fresh sperm made from stem cells that keep dividing. So sperm never face the forty-year-old-glue problem.

The trade-off is the photocopier. Every division carries a tiny chance of a copying error, and the errors add up over the years. A large Icelandic study found that each extra year of a father’s age adds about two new genetic changes to his child’s DNA5. The researchers say this helps explain why a father’s age matters for conditions such as autism and schizophrenia5.

For the number of chromosomes, though, eggs are the main story: more than 90 percent of extra-chromosome conditions trace back to the egg4.

In short, eggs hold on to decades-old glue, and sperm run a decades-long photocopier. Each ages in its own way.

Almost unique in the animal kingdom

Plenty of animals become less fertile near the end of their lives. But humans are unusual: we stop being fertile and then keep living for decades.

Among animals with backbones, the only others known to do this routinely are killer whales and short-finned pilot whales1. Why we evolved this way is still a mystery. Scientists have plenty of theories, but no firm answer.

The takeaway

Your eggs have been with you longer than almost anything else in your body. They were made inside your mother while she was inside your grandmother. They’ve waited decades, and most quietly bowed out without ever being called on. The countdown ends in menopause.

It’s not a design flaw. It’s one of the oldest and strangest stories your body carries.

If the countdown feels like it’s speeding up, our perimenopause check takes three minutes.


Sources

  1. The Menopause Society (formerly North American Menopause Society). Menopause Practice: A Clinician’s Guide, 6th edition, 2019. Chapter 1, “Stages of Reproductive Aging” and “Physiology of the Menopause Transition”; Chapter 5, “Decline in Fertility With Reproductive Aging.” Egg counts by age, AMH as the brake on follicle recruitment, dizygotic twinning with age, donor-egg pregnancy rates, mitochondrial decline, and post-reproductive lifespan in killer whales and short-finned pilot whales. https://menopause.org/professional-resources/menopause-practice-textbook
  2. Tsutsumi M, Fujiwara R, Nishizawa H, et al. Age-related decrease of meiotic cohesins in human oocytes. PLOS One 2014;9(5):e96710. Cohesin proteins REC8 and SMC1B “were found to be decreased in women aged 40 and over compared with those aged around 20 years”; per-oocyte reductions of 24 and 38 percent. https://doi.org/10.1371/journal.pone.0096710
  3. Duncan FE, Hornick JE, Lampson MA, et al. Chromosome cohesion decreases in human eggs with advanced maternal age. Aging Cell 2012;11(6):1121–1124. Distance between sister kinetochores increases with maternal age in human eggs, consistent with loss of cohesion. https://doi.org/10.1111/j.1474-9726.2012.00866.x
  4. Jessberger R. Age-related aneuploidy through cohesion exhaustion. EMBO Reports 2012;13(6):539–546. Cohesin is loaded in fetal oocytes and not replenished; its exhaustion over decades as a leading explanation for maternal-age aneuploidy; more than 90 percent of trisomies are maternal in origin. https://doi.org/10.1038/embor.2012.54
  5. Kong A, Frigge ML, Masson G, et al. Rate of de novo mutations and the importance of father’s age to disease risk. Nature 2012;488:471–475. 78 Icelandic parent-offspring trios; about two additional de novo mutations per year of paternal age; discussed in relation to autism and schizophrenia risk. https://doi.org/10.1038/nature11396